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These B2M-CuS NPs are specifically targeted to induce apoptosis in senescent chondrocytes while showing no toxicity toward normal chondrocytes

With respect to neurotrophic signaling and neuroplasticity, all three agents enhance brain-derived neurotrophic factor (BDNF)-related pathways, but through distinct biological routes

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GHK-Cu contributes ECM-remodeling, collagen and glycosaminoglycan synthesis, and broad gene-expression modulation through copper-mediated signaling

[DOI] [PubMed] [Google Scholar] Hudson DM, Archer M, King KB, Eyre DR (2018) Glycation of type I collagen selectively targets the same helical domain lysine sites as lysyl oxidase-mediated cross-linking