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Disorders of metabotropic glutamate (mGlu) receptors and neurotrophins have significant negative effects on neuroplasticity, leading to depression, but LAC regulate brain type 2 metabotropic glutamate (mGlu2) receptors and N-methyl-D-aspartic acid (NMDA) receptors to induce antidepressant effect (8385)

These differ chemically from archiestrogens (naturally occurring) produced by living organisms (7, 8)

Currently, therapies rely largely on pharmacological agents that curb inflammation by targeting immunoregulatory pathways, including Janus Kinase (JAK) inhibitors and other intracellular tyrosine kinases [5, 6]

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In contrast, CDIPT was identified only at cut-off values of 80% or higher for maximal biomass production rate, but not at more stringent thresholds (Fig