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7.3 Metabolic evasion AML cells often adapt metabolically to resist ferroptosis by upregulating NRF2, GPX4, and glutathione pathwayssimultaneous targeting of NRF2 and GPX4 restores ferroptosis sensitivity in vitro [26, 38, 122]

However, clinical application of these enzymes is usually hindered by common problems including allergy to these foreign proteins, off-target cytotoxicity, short half-life and rapidly emerging chemoresistance

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