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Cell Death Discov 2(1):16070

Dysregulated AMPK activitycharacterized by reduced phosphorylationis a hallmark of NAFLD pathogenesis, driving excessive de novo lipogenesis, impaired fatty acid oxidation, inflammasome activation, and mitochondrial dysfunction, all of which exacerbate hepatic steatosis and injury (Lv et al., 2024

For HIV patients in part due to the cocktail of medications theyre often prescribed have increased deposits of visceral fat

Studies have demonstrated that agonists of these receptors reduce serum bilirubin and increase urinary excretion of glucuronidated products in cholestatic models [55,56,57]

10.1186/S12944-020-01273-Z [DOI] [PMC free article] [PubMed] [Google Scholar] Temporelli, P