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Although initial studies suggested that GSH depletion was only a byproduct of oxidative stress generated during cell death, recent discoveries suggest that GSH depletion and post-translational modifications of proteins through glutathionylation are critical regulators of apoptosis

10.1177/1945892419878315 Am J Rhinol Allergy

6h, i, and DEN/HFD model in Supplementary Fig

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