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This sarcopenia-like phenotype arises from a combination of disuse, altered neuromuscular recruitment patterns, systemic inflammation, and age-related muscle loss (Shorter et al., 2019)

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Moreover, metabolites derived from the gut microbiota, such as SCFAs, secondary bile acids, triethylamine, and LPS, may mediate the regulation of host lipid metabolism and possess anti-obesity potential [58, 59]

Advantageously, the equine innate immune system and its antimicrobial peptides show some remarkable features offering particular chances, for example the high number of -defensins in the intestine

Regulation of cell proliferation and apoptosis: Glutathione depletion is a signal for apoptosis (programmed cell death), while adequate levels support normal cell proliferation [2][5]