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In apoptosis, -sitosterol upregulates pro-apoptotic factors such as p53 and Bax, while downregulating anti-apoptotic proteins such as Bcl-2, leading to an increase in mitochondrial outer membrane permeability and the activation of key apoptotic enzyme cascades such as caspase-9 and 3, which induces programmed cell death (83)

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At clinical doses, the peptide activates collagen synthesis, accelerates cell turnover, and modulates the inflammatory response across multiple tissue types

When hearing loss occurs at a specific frequency, the brain may generate a sound to compensate for the missing signal