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FPSZM1, a bloodbrainbarrierpermeable RAGE inhibitor, significantly attenuates HMGB1-mediated pulmonary inflammation and tissue injury in murine emphysema models, illustrating proof of concept for its use in fibrotic lung disease (159)

This effect is associated with the activation of chemoprotective mechanisms, which involves increasing white blood cell and lymphocyte levels, as well as restoring TNF-, INF-, and IL-10 levels

This is likely due to the longer chain fatty diacid promoting a tighter binding with serum albumin, thereby protecting the lipid-modified stapled peptide from esterase degradation