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W.et al (1997)

Alternatively, the lingering low level of hypoxia (1) following the acute COVID-19 infection may also contribute to lower ATP levels

1k), suggesting that CAR T cells from LTR preferentially shunt HK glycolytic metabolites to the pentose phosphate pathway (PPP) rather than glycolysis, and by CPT1A could convert long-chain fatty acids into acylcarnitines for shuttling into the mitochondrial matrix for subsequent oxidation, previously associated with increased basal and maximal mitochondrial respiration 5,6,7,8

More recently, hypoxia-inducible factor prolyl hydroxylase inhibitors (HIF-PHIs) have received clinical approval for their ability to promote hematopoiesis through enhancing endogenous EPO production (McCallum et al., 2022)