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IGF-1 regulates dopamine release from these neurons, and the absence of IGF-1 in dopaminergic neurons decreases dopamine levels in the striatum, leading to deficits in dopamine neuron firing, spontaneous locomotion, and exploratory and learning behaviors (53)

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In microglia, FMN suppresses KMT2B expression, thereby downregulating TNFR1/NF-B activity and curbing the release of pro-inflammatory cytokines like IL-1 and TNF- (10, 66)

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