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The amplified plaque inhibition efficacy associated with P-Mn/TI@mHSA aligned with its enhanced atherosclerosis binding and plaque-penetrating capabilities demonstrated above, which facilitated the delivery of the Mn 3 O 4 - and TI-loaded protein nanoclusters into plaque macrophage cells for ROS scavenging and TRAF6 inhibition

Within this framework, ferroptosis is not merely a downstream consequence of A/Tau pathology but also an upstream driver and a sustained propagator throughout the disease continuum

Combined with the systemic anti-aging effects on gene expression, this peptide delivers value beyond just skin appearance