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IL-1 antibody treatment markedly reduced macrophage abundance and IL-6 expression in the tissues (Fig

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doi:10.1089/thy.2020.0139

Hepatic effects that are considered adverse include death, abnormal clinical chemistry parameters, obvious functional organ impairment, DNA and mitochondrial alteration, glutathione depletion, hepatocyte necrosis, enhanced replicative DNA synthesis, generation of reactive oxygen species, and increased peroxisome proliferation (in rodents) (Williams and Iatropoulos 2002)

Understanding the context-dependent relationship between the autophagy pathway and cell death and the key regulator of this interplay may establish new approaches for the treatment of human diseases