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Moreover, other molecules that are overexpressed in DS patients and are also related with cardiac hypertrophy could unveil similar questions: Are the elevated expression levels of DYRK1A, RCAN3 and DSCAM-AS1 involved in the cardiac hypertrophic response prevention observed in DS individuals

[8] GHK-Cu and Oxidative Stress One research hypothesis suggests that GHK may contribute to reducing the release of iron from ferritin, which catalyzes lipid peroxidation

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