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AMD primarily oxidative damage and inflammation, choose antioxidant and anti-inflammatory peptides (GHK-Cu, BPC-157)

However, the early observation by Bladanzi and co-workers in which both ghrelin and des-acyl ghrelin inhibited apoptosis in H9c2 cardiomyocytes which do not express GHSR1ainaugurated the alternative line, sustaining that prosurvival pathways may be triggered and functionally activated through a GHSR-independent pathway (Baldanzi et al., 2002)