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Cellular players in kidney fibrosis Fibrosis progression is driven by the interplay and transformation of several key cell types: Tubular epithelial cells (TECs) TECs are deeply involved in chronic kidney damage by activating a variety of pro-fibrotic pathways [e.g., Wnt/-catenin, Sonic Hedgehog (Shh) signaling, and TGF-] [6,7,8] and undergoing a partial Epithelial-to-Mesenchymal Transition (pEMT)

The latter of which can attack any compound near its production

LR3-IGF-1 bypasses the GH-axis and directly engages the IGF-1 receptor producing IGF-1-receptor effects without the broader pleiotropic GH effects

Z.XiaT.LinJ

Activation of ghrelin receptors in the hypothalamus and pituitary integrates endocrine output with metabolic regulation pathways