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Neuron-glia interactions in the pathophysiology of epilepsy

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They regulate critical biological functions like healing, inflammation, immune response, and tissue regeneration

A mitochondria-targeted covalent organic framework (COF) prodrug that is selectively activated by high intramitochondrial GSH levels was shown to trigger catalase collapse within the organelle, thereby amplifying local chemodynamic therapy (CDT) through enhanced ROS accumulation and mitochondrial dysfunction