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Higher levels of NFKB-associated cytokines (IL1B, IL8, TNF, GM-CSF) were detected in HepG2 ATP7B-KO cells compared to HepG2 cells (Fig

This metabolic reprogramming led to mitochondrial membrane depolarization, increased reactive oxygen species generation, induction of apoptosis, and cell-cycle arrest at the G 2 phase

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