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Patients with various degrees of TBI may suffer from a long period of secondary injury, which plays a key role in determining the final severity and recovery of nerve injury after TBI ( FIGURE 1 Inflammation Neuroinflammation is a key pathophysiological mechanism of secondary injury, as it can produce neurotoxic effects on neurons through oxidative stress, apoptosis, and excitotoxicity (Sulhan et al., 2020)

PubChem Compound Summary for BPC-157 (CID 9941957): molecular formula, molecular weight, and sequence

Gordon, P

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Toxicol In Vitro 30(1 Pt B):429437 Hong F, Freeman ML, Liebler DC (2005) Identification of sensor cysteines in human Keap1 modified by the cancer chemopreventive agent sulforaphane