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therefore, the therapeutic use of SOD and SOD-mimetics (e.g., Mangafodipir ) may prove beneficial in PASC recovery 565,569

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Masino SA, Rho JM

The molecular mechanisms proposed to explain how ROS could specifically mediate brain damage are the following: (1) The lower levels of antioxidants can promote increased activity of lipoxygenase in CNS stimulating leukotriene production, thereby increasing the immunoinflammatory processes in the cerebral cortex

More concerning manifestations include elevated thyroid antibodies detected through blood tests, indicating potential autoimmune thyroid conditions