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Select heterozygous Keap1 mutations have a dominant-negative effect on wild-type Keap1 in vivo

Co-authors with New and Kearney are Brian L.P

However, it remains to be determined whether its beneficial effects are primarily mediated via the inhibition of direct PPAR target genes, such as CD36, or through the PPAR-mediated repression of proinflammatory transcription factors, such as NF-kB

thus, GCL is considered the rate-limiting enzyme [6]

Isha Narang Senior Consultant Dermatologist & Hair Transplant Surgeon 14+ years experience Accomplished Consultant Dermatologist with extensive international experience in general and pediatric dermatology