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Scientific reports 12(1), 2022

The restoration of mTOR signaling with Fer1 rescue treatment in the context of RSL3 effects, and the genetic knockdown of GPX4, reaffirm our in vitro findings that RSL3-mediated induction of ferroptosis, mTOR pathway suppression, DNA damage and failure of DNA repair, and autophagy are GPX4-dependent processes in thyroid cancer cells

However, this association does not imply causationelevated B12 often serves as a biomarker for serious underlying disease rather than being directly harmful

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