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Maki, 2009

This finding triggered the amyloid hypothesis, which is the idea that APP cleavage products, such as A42, are the fundamental cause of AD, eventually leading to synaptic dysfunction, gliosis, neuronal loss, and tau pathology (Hardy, 2009)

Genomically informed patient stratification Given the large variability in BM treatment responses, there remains a significant need for clinically relevant biomarkers to prospectively stratify patients to the optimal treatment strategy

Phase 3 TRIUMPH-4 data showed 9mg achieved 26.4% weight loss versus 28.7% at 12mg over 68 weeks