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While the dithiol mechanism for EcGrx1 and EcRNR or EcPR can be explained by specific protein-protein interactions (Fig

Endothelial dysfunction as an early sign of atherosclerosis

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[5] The palmitoylation cycles of a wide array of enzymes have been characterized in the past few years, including H-Ras, Gs, the 2-adrenergic receptor, and endothelial nitric oxide synthase (eNOS)

Selective mitochondrial autophagy, or mitophagy, as a targeted defense against oxidative stress, mitochondrial dysfunction, and aging