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Its defining structural feature is an N-terminal trans-3-hexenoic acid modification, a short unsaturated fatty acid attached to the amino terminus that shields the peptide from rapid enzymatic degradation by dipeptidyl peptidase IV (DPP-IV)

Sadeer, N

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Inflammatory lung diseases are often characterized by chronic inflammation and an imbalance between oxidants and antioxidants, a primary cause of cellular damage [12]