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thigh) has a minimal impact on the GHK-Cu half life

The specific mechanism by which CitAgNPs induce oxidative stress involves potential cleavage of the cell wall and subsequent entry into the cell, where the released silver ions may interact with cellular components, disrupting redox balance

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NMDA antagonism exacerbates repetitive behaviors in the D1CT-7 mice, described above (McGrath et al., 2000)

Previous studies have also reported that feeding Apoe (/) mice with tocopherols and tocotrienols downregulated the expression of CD36 and upregulated the transcriptional activity of LXR , ABCA1 , and peroxisome proliferator-activated receptor gamma ( PPAR ) (Tang et al., 2014)