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Taken together, these pieces of evidence suggest that the biosynthesis rate of glutathione might be involved in nephrotoxicity by increasing cellular retention of Hg, altering the redox balance and leading to cytotoxicity 46,47 , possibly via mitochondria dysfunction by altering the ratio of reduced (GSH) to oxidized (GSSG) glutathione in this organelle 48,49

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Other research suggests that B5, pantothenic acid, and B12, cobalamin, are important for maintaining glutathione levels

Abstract Lack of effective drugs for acute kidney injury (AKI) grades 12 is a crucial challenge in clinic