Since Asp in the blood cannot directly pass through the blood-brain barrier, the development of nerve cells depends on its synthesisation in the brain
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We found that wild-type (WT) ACTL6A and the DNBL, CC, and NBC ACTL6A mutants reversed the inhibition of cell proliferation and the reduced GCLC protein expression induced by knocking down ACTL6A expression, but the ACTL6A HR-deleted mutant did not show this restorative effect (Fig
It can also be found in bodily fluids such as plasma, saliva, and urine
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