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Reported cellular effects include enhanced fibroblast proliferation, collagen production modulation, and improved organization of extracellular matrix components at repair sites

To prevent the excessive antigen stimulation response, cancer cells suppress the T-cell activity by binding of PD-1 to ligand on cancer cells such as PDL-1

When the gut epithelial barrier is disrupted, lipopolysaccharide (LPS) and other detrimental microbial products enter the host's circulation, leading to the activation of systemic inflammation [2]