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This receptor upregulation combines with BPC-157's activation of the Src-Caveolin-1-eNOS signaling cascade, which increases nitric oxide production by 1.35-fold and proves essential for the peptide's pro-migratory effects on endothelial cells

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This structural modification significantly extends its half-life and enhances resistance to proteolytic degradation, thereby enabling prolonged biological activity in vivo

Experimental Biology and Medicine, 245(15), 14171425

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