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In this context, targeting QA, or the enzymatic steps that govern the balance of KP metabolites, may represent a more tractable therapeutic strategy, allowing attenuation of NMDAR-driven excitotoxicity without disrupting 7nAChR-dependent cognitive processes

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Studies have shown that this epithelial damage is in part due to the inability to mitigate reactive oxygen species (ROS)

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Changes in the diurnal rhythms of cortisol, melatonin, and testosterone after 2, 4, and 7 consecutive night shifts in male police officers