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The redox couple GSH/GSSG represents a critical determinant of the cells antioxidant defense capacity [12]

Presuming this is true, we would expect to observe no differences in measured free GSH between the deproteinized heparinized plasma and deproteinized EDTA-treated plasma stored at 80 o C from the same subjects

Studies have suggested that increased susceptibility to KBD in selenium-deficient populations might result from a reduced antioxidant protection associated with polymorphisms in GPX genes (60, 61), but polymorphisms in other genes have also been implicated (62)

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These reactions represent either deep tissue abscess formation or delayed Type IV hypersensitivity, both of which contraindicate continued glutathione use without provider reassessment