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Traumatic brain injury (TBI) is characterized by both the primary damage resulting from physical disruption of neural and vascular structures and the early emergence of secondary pathogenic events, which collectively contribute to neurologic deficits (Andriessen et al., 2010

A six-year-old boy with hypohydrotic ectodermal dysplasia, immunodeficiency and a novel mutation in the nuclear factor kappa B essential modulator (NEMO) gene

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