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This work suggests that HGF-MET may also protect from parthanatos, a form of caspase-independent regulated cell death occurring especially in neurons and driven by PARP1 hyperactivation (Galluzzi et al., 2018)

[9] That mixed result is worth stating rather than smoothing over, because it illustrates a real feature of the preclinical record: effects vary by model, species, formulation, and readout, and the literature is not a monotone chorus of positive findings

Finally, we aimed to confirm endogenous S 0 generation more directly, and independently of roGFP2-based measurements