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Key molecular mechanisms encompass oxidative modification of critical proteins leading to dysfunction [361], membrane lipid peroxidation disrupting organelle integrity [362], mtDNA damage causing mitochondrial dysfunction [363], and depletion of key antioxidant molecules [364]

This suggests DSIP functions as a normalizing agent rather than a universal sedative

10.1016/j.tox.2006.06.008 38

MZ: Conceptualization, Funding acquisition, Writingreview and editing

GA also alleviates oxidative stress via Pyruvate Kinase M2-mediated mechanisms and inflammation, reducing hepatocyte apoptosis and thus mitigating TGT (anti-rheumatic)-induced acute liver injury (Wang Q