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Antioxidation activities of low-molecular-weight gelatin hydrolysate isolated from the sea cucumber Stichopus japonicus
The susceptibility of cancer cells to ferroptotic death is determined by the antagonism between the cellular metabolic activities that trigger lipid oxidation and the cellular antioxidant systems that counteract it [34]
The primers used are listed in the Supplementary Table 2
which has respective convenience over SLN as NLC comprises of liquid lipid in addition to solid lipid which provides much stability and flexibility to nanoparticles, provide higher drug loading capacity, etc
Combined interventions targeting MPC dysfunction, ROS buffering systems such as the Nrf2GSH axis, or mitophagy pathways including PINK1, may disrupt the self-sustaining signalmetabolism circuit and offer a strategy to delay or reverse acquired therapeutic resistance
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