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Under pathological conditions characterized by deficient mitophagy, pharmacological agents such as rapamycin or aai berry restore mitochondrial turnover, thereby promoting apoptosis and reducing angiogenesis in lesions reliant on AKT/mTOR signaling (6668)

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Injectable BPC-157 enters the bloodstream, providing systemic delivery and often faster tissue repair, though it may not be as localized to the gut unless carefully targeted

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