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Relative to the wild-type enzyme, mutation of residue 15 to serine or alanine in UGT74F2 did not lessen the specific activity for SGE formation, but did increase the formation of SAG

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Neuron 18 , 327338 (1997)

MihlanMBlomAMKupreishviliKLauerNStelznerKBergstrmFet alMonomeric C-reactive protein modulates classic complement activation on necrotic cells

The pathogenesis of OA is highly complex, involving interactions among multiple factors such as abnormal mechanical loading, age-related metabolic changes, persistent inflammatory responses, dysregulated extracellular matrix(ECM) metabolism, and genetic susceptibility