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Unlike sepsis, where treatments are primarily directed against LPS-induced inflammatory responses (e.g., anti-endotoxin antibodies), SAP-induced glycocalyx damage is predominantly driven by pancreatic enzyme activation (e.g., trypsin) and intestinal endotoxin release

Andreja Trpkovic, Ivana Resanovic, Julijana Stanimirovic, Djordje Radak, Shaker A

Szerencsre a kutatsok azt is kimutatjk, hogy bizonyos gygynvnyek, tpanyagok, s a kofaktoraik aktivljk a glutation reduktzt s a reduklt glutation szintzist

2004, pp

In fact, LP can persist in many parts of the brain, including the brainstem, for decades without causing any observable degeneration or cell death