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Microbiome and chronic wound infections: mechanisms and interventions Building upon the clinical challenges outlined in the introduction, this section explores the central role of the microbiome in chronic infections and its disruption in wound healing

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In this setting, any perturbation that lowers intracellular GSH to an as yet unknown threshold could eventually induce cell death and, in all likelihood, ferroptosis, but also significantly increase expression and secretion of numerous T2-associated proteins

Quantitative proteomics identifies NCOA4 as the cargo receptor mediating ferritinophagy