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The consequences of IGF-1 deficiency on skeletal growth become apparent by E13.5, with mutant mice having approximately 90% of the body weight of normal controls

GO analysis further showed that uridine supplementation restored biological processes in aged oocytes, including oxidative stress responses, iron ion homeostasis regulation, lipid metabolism, oxidative phosphorylation, cell cycle regulation, and DNA repair (Fig

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When stress persists, the -cell gradually retreats and converts into a dedifferentiated pancreatic progenitor, which might be more resistant to high-glucose/ROS challenges 22,29 and thus survives

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