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Some studies suggest interaction with inflammatory signaling markers in experimental models, though conclusions remain preliminary

However, GSH acts like a helper cell to restore antioxidants back to their reduced forms and allow them to continue to fight free radicals 6

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The need for diagnosis, treatment and prevention of AITD is also due to the need to prevent concomitant diseases, including disorders of the cardiovascular system, hypercholesterolemia, atrial fibrillation, encephalopathy, others autoimmune diseases ( Cellular and humoral immunity in the pathogenesis of AITD In addition to genetic, cellular and humoral immune mechanisms, loss of immune tolerance to thyroid autoantigens is the basis for the development of AITD