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DNA damage triggers a prolonged p53-dependent G1 arrest and long-term induction of Cip1 in normal human fibroblasts

At the end of the experimental period, the DZN-treated group had an average weight of 37.67 g, which is significantly less than that of the control group (41.78 g)

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This is supported by compelling evidences that IGF1 replacement therapy is able to restore mitochondrial dysfunction observed in untreated cirrhotic rats and in IGF1 partial deficiency mice (Perez et al., 2008

Faseb j 33(11):1228812298