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One study, which lasted a month, reported that GHK-Cu might stimulate type 1 collagen production in clinical settings

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Both variants had not previously been described and were classified as pathogenic according to the American College of Medical Genetics (ACMG) guidelines [13]

While its exact mechanisms are still being elucidated, research suggests it may work through modulation of calcium channels, influence on cyclic nucleotide levels, and interaction with various neurotransmitter receptors