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To test this, GPx1 expression was partially knocked down in macrophages from healthy volunteers and this resulted in increased H 2 O 2 -induced cell death and lipid peroxidation without H 2 O 2 treatment, implying that GPx1 is important in the homeostatic maintenance of cellular lipid redox

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During the Open-Period proposal, submissions must be made through the Defense SBIR/STTR Innovation Portal or DSIP

Western blot HepG2 cells were treated for 24 or 48 h with selected drugs and were harvested in the following buffer: 150 mM NaCl, 1% Igepal, 50 mM TrisHCl pH 7.4 with Protease inhibitor cocktail (Roche)